It is a repeating trend in all medical research. However enough does turn out to work in humans that we eventually make useful progress.

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wk_end 3 hours ago | parent [-]

In general, sure, but in this specific instance (treating Alzheimer's by clearing amyloid-beta) it's been shown over and over again to not work in humans.

	▲	russfink 12 minutes ago \| parent [-]
		I’m kinda new to this - so what you’re saying is the mouse model induces beta-amyliods directly, rather than finding ways to give mice Alzheimer’s, whereas the human tests are for humans that have Alzheimer’s? Meaning we aren’t doing any tests of simply stimulating BA growth in humans?